TWATTER: Hacking away at the India narrative today. Back on this account at present ⬇️
(@robinmonotti2): https://twitter.com/robinmonotti2?s=09

LETTER TEMPLATES ✒

http://miriaf.co.uk/category/letters/

INDIA: After a long consideration of hundreds of articles & witness statements, I am calling it today: we are seeing a textbook case of mass vaccine antibody-dependent enhancement. Vaccinated people, especially those going for their first dose therefore by definition the least protected & most fearful and in a state of panic and stress, which already increases cytokines even before the disease, are being exposed to the wild virus in the same hospitals they are vaccinated in or through contaminated water, as SARSCoV2 can replicate in bacteria (Brogna et al, 2020). Subsequently a small percentage of them may develop a vaccine ADE enhanced form of Covid19, which also enhances symptoms & therefore enhances spread to their family members, some of which may also develop vaccine ADE. The cycle continues, more vaccinations, more v-ADE, more infections, more deaths. The only way to stop this is to halt vaccinations immediately, and revert to early treatment. The Ziverdo Kit is a good example of an effective combined treatment. Remdesivir is comparatively not as effective & may even exacerbate vaccine antibody dependent enhancement.

"ANTIBODY-DEPENDENT ENHANCEMENT - Why C19 injections may increase deaths when faced with wild virus: "Antibodies can worsen disease in coronavirus infection in animals & possibly humans..Subneutralizing antibodies can promote viral entry into cells"
https://www.sciencedirect.com/science/article/pii/S2590136220300231

Remember "Your [natural exposure] Immune System [BOTH antibodies & T-cells] Evolves to Fight Coronavirus Variants" & mutants. Don't fear them if you have natural immunity.
https://www.scientificamerican.com/article/your-immune-system-evolves-to-fight-coronavirus-variants/

This isn’t a new paper, but is an excellent review of immune responses to viral infection of that lung:
https://www.researchgate.net/profile/David-Woodland-2/publication/23422086_Immunity_to_Respiratory_Viruses/links/00b49528fb46fa39c4000000/Immunity-to-Respiratory-Viruses.pdf
Immunology is truly amazing! I knew that due to gene shuffling during the time the cells, which will later differentiate into your T-cells, already each possess one of millions of potential T-cell receptors (TCR) which will perfectly match up to a piece of an invading virus.
How cool is that? And because the range of antigens you might encounter through your life is potentially very diverse, you’d rightly not want to give any of them up. You might need them, to protect you against a virus variant 🤭
It’s hard to read this review without concluding, as I’ve said, that T cells are critical to pulmonary host defence v respiratory virus infection. The clearest evidence for a role for antibodies in virus host defence of the lung comes from study of secretory IgA, not the circulating varieties which some obsess about. You’d want all the tools in a nearby spot, but you definitely don’t want to try this without T-cells.

A major limitation of studies is that almost all of them are done in mice & we know of a number of important differences in cytokines / chemokines between mice & humans, as well as anatomical differences. Worst of all, the lab mice are so clean that their ‘naive’ immune responses almost certainly don’t accurately represent what happens in our ‘experienced’ airways.
For me though, it’s reinforced my confidence that memory recall immune responses in airways to uncomplicated virus infections are substantially effected by T-cells, an expanded repertoire of which easily detects infection by related viruses (especially including close-in variants, which ALL the “scariants / same-iants” of SARS-CoV-2 are).
Enjoy!
Mike
Cheers
Mike

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TOOLKIT OF CONTROL

Investigative journalist Harry Vox's prophetic interview from 2014 where he warns about public health crises being used as tools of control.

"If the ruling class ever saw wide scale civil unrest, you'd see an ebola outbreak in America right away"


https://m.youtube.com/watch?v=wuOxG-rnj30

https://m.youtube.com/watch?v=I21Vt6rParw
Robin,this is worth a watch, it's from 2015 by a guy called Dr Graham Downing, it might be worth you or Mike trying to contact him. He seems to know alot of what's going on , it could be a talk in 2021 it's that appropriate to now!!

This reinforces my view that inducing the spike protein through injections may cause vascular damage which may have been prevented by a localised lung T-cell response.

"CD8+ T cells with an intraepithelial phenotype upregulate cytotoxic function upon influenza infection in human lung

We previously published that respiratory virus-specific T cells reside in the human lung, rather than in the peripheral blood (10), but their location within the lung had not been studied. The presence of virus-specific memory cells at the viral entrance site is important, since this can facilitate local antigen presentation, which could enable these virus-specific T cells to directly exert their effector function, without migration to the lymph node being necessary (36). This way the viral load and epithelial damage can be reduced before the recruitment of memory cells from the circulation and lymphoid tissue has taken place, thereby substantially reducing disease duration and pathology. In addition, immunopathology may be limited, as the initially reduced viral load will lead to a reduced degree of immune activation (37).

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3104744/

GULLAIN-BARRE SYNDROME POST VAXX ⚠️
"The COVID-19 virus has spike (S) proteins on its outer surface used for cell entry and trigger for disease. The ChAdOx1 vaccine is a ‘monovalent vaccine composed of a single recombinant, replication-deficient chimpanzee adenovirus (ChAdOx1) vector that codes for the S glycoprotein of SARS-CoV-2 (ChAdOx1-S (recombinant))’.13 On vaccination, the vector enters cells stimulating the S glycoprotein of COVID-19, which is then exported to the cell surface, provoking the production of antibodies and T cells. This process theoretically provides immunity for a further exposure to COVID-19 in the future, the duration of which is yet to be established. However, in selected patients, we postulate that this mechanism may be associated with a molecular mimicry response, therefore leading to the assembly of antibodies against neuronal myelin sheaths and resulting in GBS.
A meta-analysis and systematic review identified the reported yearly incidence of GBS within the general population of Europe and North America to be 0.81–1.89 (median 1.11)/100 000 people.20 Furthermore, the MHRA stated that 6 patients from 42 917 developed GBS following the ChAdOx1 vaccine, with no further information available.21 Additionally, one case of GBS was reported 2 weeks following the patient’s first dose of the BNT162b2 COVID-19 vaccine being administered.22 Similar to our case, the patient presented 2 weeks after their first dose of the vaccine, with a neurological sequelae consisting of areflexia, decreased pinprick sensation in the lower limbs and abnormal gait. The patient was diagnosed with GBS following elevated protein in the lumbar puncture and an MRI spine demonstrating enhanced cauda equina nerve roots. The team also treated the patient with 5 days of IVIgs, with neurological improvement following the third dose and subsequent discharge to an acute rehabilitation unit. In contrast, our patient did not show drastic improvement with IVIgs and the neurological deficit remained static. Research suggests that 40% of patients who have the standardised treatment of IVIg or plasma exchange do not show immediate recovery within the first 4 weeks.23"

https://casereports.bmj.com/content/14/4/e242956

⬆️ Please like this one and let's save some 🐑 from next December's likely ADE.

"Guillain-Barre syndrome (GBS) is an acute, autoimmune demyelinating polyneuropathy characterised by progressive, proximal spreading muscle weakness. This is often self-resolving; however, in severe cases, it can be life-threatening secondary to respiratory muscle compromise, requiring mechanical ventilation.5 There have been a few reported cases documenting a possible link between COVID-19 and neurological manifestations, such as GBS, although an uncommon association.6–9 We describe a fascinating case of a 37-year-old man who was diagnosed with GBS 3 weeks post the first dose of the ChAdOx1 vaccine, in the absence of any other triggering factors."

https://casereports.bmj.com/content/14/4/e242956

Dr Mike Williams for @ukcolumn
"Conclusion: The data clearly demonstrate that the last thing you would ever want to do is make a vaccine that produces a spike protein."
https://www.ukcolumn.org/article/clotting-and-covid-science

"The World Economic Forum recently ran a Livestream with Interpol, Cloudflare, and a cybersecurity firm to discuss the upcoming cyber pandemic agenda, which many believe will be their next step to justify creating a Digital Citizenship to be able to even use the internet. Cloudflare’s COO who is advises on digital identity/citizenship for internet users. You want to associate everything you do on the Internet with a digital ID. Here is Cloudfare’s COO Michelle Zatlyn casually talking about 100% security which will be used to block anyone in this cancel culture that they disagree with."

“The doctors say it’s hard to prove that it’s linked to the covid jab and that the infection could have been there for some time, but it’s strange that I became ill for weeks on the night of the vaccine." https://www.stamfordmercury.co.uk/news/amp/man-lost-leg-weeks-after-covid-vaccination-9198171/