Robin Monotti
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2 of the 4 authors of the study work at the European Commission. Another works at an Italian medical research facility called Craniomed: http://www.craniomed.it/
Robin Monotti
Here is the Italian-EU funded scientific study indicating SARSCoV2 replicates in bacteria, not only human cells, and that is why antibiotics work and these vaccines will not: "SUMMARY: Scientific evidence for the involvement of human microbiota in the development…
THIS IS HUGE.
VACCINES REDUNDANT.
VACCINE PASSPORTS USELESS. IT'S ALL OVER.
THEIR STRATEGY HAS LOST ALL ITS BASIC ASSUMPTIONS TO REAL SCIENTIFIC EVIDENCE. THE VIRUS REPLICATES IN BACTERIA! ALL WE EVER NEEDED WERE ANTIBIOTICS!!!
This is where they conducted the study, it looks like a serious website for a medical research facility which is clearly funded or close to the European Commission as 2 authors of the study work for the European Commission.

http://www.craniomed.it/
Forwarded from Mike Yeadon
Interesting! Ok, maybe the virus replicated in bacteria. I know some viruses do. Viruses commandeer the replication apparatus of anything which itself makes generic copies of itself.

But are we sure what it is they’re measuring?

I’ve seen some work showing that the some of the PCR probes & primers in some of the kits do match sections of bacterial genomes.

I’d like them to additionally determine concentrations of antigen as measured in lateral flow (immobilised antibody) tests.

Coin toss whether they pick up increases or not!

Will read others comments with interest. This looks to be a rather specialised field.

Mike
“Sars Cov 2 is also a bacteriophage virus. It means that it enters the bacteria and replicates its RNA from there as well. We finally have the scientific evidence, complete with photos of the virus colonizing the bacterium." Carlo Brogna, Craniomed, Italy.
"The discovery that Sars Cov 2 is a bacterial virus has enormous consequences. The risk among vaccinated people of fatal enhancemeny of the disease or ADE. The phenomenon of vaccine resistance causing the spread of more dangerous and contagious mutantions of the virus" Dr Loretta Bolgan, Italy
https://scenarieconomici.it/moltissimo-di-cio-che-ci-dicono-su-virus-e-vaccini-e-sbagliato-il-resto-e-nascosto/
See what they were trying to distract us from now? The whole house of vaccine ID cards is premised on bad science, we can blow it down if we all blow at the same time in the right direction.
SARSCoV2 "injects its own genetic content into the bacterium and from there it is replicated. We have absolute and indisputable evidence of this with the many experimental tests we have performed. We are also collecting electron microscope photos. Let me explain. A virus normally attacks our cells, namely the epithelium and mucous membranes. It tries to enter and replicate itself and this induces an immune response in the host. It triggers a series of biochemical events to which the body responds with inflammation proteins. But above our cells there is the microbiome, or a layer of "good" bacteria that live with us. In the case of a bacteriophage virus, it does not pass without first interfacing with the microbiome, but also enters the bacteria. We were tied to the classic concept that the problem is that phages are usually ten times smaller than a coronavirus! Diphtheria, for example, is also a disease caused by a bacterium that produces toxins because it has been colonized by a phage virus."

https://www.affaritaliani.it/coronavirus/vaccino-attuale-non-sufficiente-ci-vuole-altro-per-debellare-il-virus-720062.html
❤1
"How did you understand this?

We at Craniomed do research on proteins. The absence of taste and smell in Covid patients was the subject of study and we used a very fine method, mass spectrometry, to find some abnormal proteins, which interfere with the nerve transmission of the two senses mentioned above. We have found such abnormal proteins in the blood and urine in people affected by COVID-19. Well, we have observed the same toxins several times in the same person but each time with different amino acids and this has made us go back to a concept of classical biology: when there are defects in the production of amino acids in the person, the origin of the problem is of type bacterial."
"Can you explain exactly how the evidence was reached that Sars Cov 2 replicates its RNA in bacteria?

I will try to explain it in a simple way. We did this by culturing bacteria from our microbiome and the virus. First, we took the fecal swab of some COVID-19 patients and cultured it for up to 7.14 and 30 days, seeing that the virus increased RNA production. Then, we took the virus and cultured it with bacteria from the fecal swab of a non-sick person, seeing that it replicated here too. Finally, we took the bacteria from the first experiment and separated them from the virus! We grew them on their own and at 30 days they were still producing virions. The evidence was clear and we were incredulous at what we observed."
SARSCoV2 "can be treated with antibiotics and avoiding some drugs but at "time zero".
Q: "What antibiotics? And also here what evidence do you have?"
A: "We tested 18 antibiotics and treated 115 symptomatic infected with previous pathologies. Azithromycin, amoxicillin, metronidazole and vancomycin work great. We gave these antibiotics and probiotics immediately and at “time zero”. The problem with this virus is toxins. Some enhance the drugs normally taken by people being treated for other previous diseases. For example, anti-inflammatories such as ibufrofen and even paracetamol cause many problems. Let's take an example. We have the rationale that bacteria produce a protein very similar to phospholipase A 2, which acts on inflammation and enhances pneumonia and coagulation of the vascular microcirculation. If we administer an anti-inflammatory NSAID we will amplify this effect. That is, we make pneumonia more severe. Increases in coagulation, neurological problems, pneumonia have resulted from this toxin-drug synergy. The toxin similar to conotoxin is the one that is most deleterious and is the one that acts on our autonomic nervous system and increases the effect of our neurotransmitter, acetylcholine, therefore all people who take drugs such as to name a few are at greater risk. ace inhibitors, beta blockers, calcium channel blockers, drugs for Parkinson's, and other similar diseases"
"What about current vaccines? Are they needed in the light of this evidence?"

"We need other vaccines, in addition to these already present. Since Sars Cov 2 also replicates in bacteria, current vaccines will have partially sufficient efficacy. For RNA vaccines that derive from gene therapies, we do not yet know if there are any long-term positive effects in terms of efficacy. Instead, the vaccine solution against toxins will certainly be taken into consideration. I clarify, they are pros for vaccines but they must be the right ones, it cannot be ignored that the virus is also a bacteriophage and that above all powerful toxins are released! Something is needed here to fight the toxins. I remember that the diphtheria vaccine did exactly that." CARLO BROGNA
See above. The logic of vaccine passports when the vaccines are even partially the "wrong" ones for such a virus is now entirely not tenable even in principle. We need to insist on this point: they are the wrong type of vaccine as they were designed by scientists who did not know the virus spreads also through bacteria. Antibiotics or bacteriostatics are more appropriate even in principle at this stage, together with any antiviral supplement such as zinc, vitamins C&D, & Ivermectin. Vaccines? Wrong design in the first place.
I am posting this once more, as it's groundbreaking:
"Scientific evidence for the involvement of human microbiota in the development of COVID-19 disease was reported recently. We elaborated these findings further and collected data on the relationship between faecal bacteria, isolated from stool from COVID-19 patients, and SARS-CoV-2. The preliminary results suggest that SARS-CoV-2 replicates in bacterial growth medium inoculated with a stool sample from an infected patient and that the replication follows bacterial growth.
These results are unexpected and when confirmed on large sample sizes hint towards novel hypotheses on the biology of SARS-CoV-2 and on the COVID-19 epidemiology.
The data reported here suggest a possible ‘bacteriophage-like’ behaviour of SARS-CoV-2, which to our knowledge was never observed or described before. The discovery of possible new modes of action of SARS-CoV-2 has far-reaching implications for the prevention and the treatment of the disease, necessitating quick sharing of even preliminary findings with the global scientific community."
https://zenodo.org/record/4088208#.YE6KnE8XadO