"In this article, we note that human host cells sensitively respond to the spike protein to elicit cell signaling. Thus, it is important to be aware that the spike protein produced by the new COVID-19 vaccines may also affect the host cells. We should monitor the long-term consequences of these vaccines carefully, especially when they are administered to otherwise healthy individuals. Further investigations on the effects of the SARS-CoV-2 spike protein on human cells and appropriate experimental animal models are warranted."

Vaccines | Free Full-Text | SARS-CoV-2 Spike Protein Elicits Cell Signaling in Human Host Cells: Implications for Possible Consequences of COVID-19 Vaccines | HTML
https://www.mdpi.com/2076-393X/9/1/36/htm

If I can be very explicit in terms of strategy, if you don't want the vaccine passport the first thing you need to do is discredit the vaccines. There are plenty of safety grounds upon which they should be discredited. That is the nail we need to be banging with all our hammers, partially because that is how people will also save their immune systems .

REMINDER: ALL CURRENT VACCINES/GENE EDITING USE THE SARSCOV2 SPIKE PROTEIN

"Vaccines that introduce the spike protein into our body to elicit virus-neutralizing antibodies.. In this article, we note that human host cells sensitively respond to the spike protein to elicit cell signaling. Thus, it is important to be aware that the spike protein produced by the new COVID-19 vaccines may also affect the host cells."

"SARS-CoV-2 Spike Protein Elicits Cell Signaling in Human Host Cells: Implications for Possible Consequences of COVID-19 Vaccines

SARS-CoV-2 Spike Protein Elicits Cell Signaling in Human Cells

It was found that the treatment of cultured primary human pulmonary artery smooth muscle cells (SMCs) or human pulmonary artery endothelial cells with the recombinant SARS-CoV-2 spike protein S1 subunit is sufficient to promote cell signaling without the rest of the viral components [21]. Furthermore, our analysis of the postmortem lung tissues of patients who died of COVID-19 has determined that these patients exhibited pulmonary vascular wall thickening, a hallmark of pulmonary arterial hypertension (PAH) [21]. Based on these results, we proposed that the SARS-CoV-2 spike protein (without the rest of the viral components) triggers cell signaling events that may promote pulmonary vascular remodeling and PAH as well as possibly other cardiovascular complications

..Kuba et al. [28] showed that the injection of mice with recombinant SARS-CoV-1 spike protein reduced the ACE2 expression and worsened the acid-induced lung injury. In mice with an acid-induced lung injury, the recombinant SARS-CoV-1 spike protein dramatically increased angiotensin II, and the angiotensin receptor inhibitor losartan attenuated the spike protein-induced enhancement of lung injury [28]. Thus, these in vivo studies demonstrated that the spike protein of SARS-CoV-1 (without the rest of the virus) reduces the ACE2 expression, increases the level of angiotensin II, and exacerbates the lung injury.

The SARS-CoV-2 spike protein without the rest of the viral components has also been shown to activate cell signaling by Patra et al. [29]. The authors reported that the full-length SARS-CoV-2 spike protein expressed by the means of transient transfection, either in the human lung alveolar epithelial cell line A549 or in the human liver epithelial cell line Huh7.5, activated NF-κB and AP-1 transcription factors as well as p38 and ERK mitogen-activated protein kinases, releasing interleukin-6. This cell signaling cascade was found to be triggered by the SARS-CoV-2 spike protein downregulating the ACE2 protein expression, subsequently activating the angiotensin II type 1 receptor [29]. These experiments using transient transfection may reflect the intracellular effects of the spike protein that could be triggered by the RNA- and viral vector-based vaccines.

These results collectively reinforce the idea that human cells are sensitively affected by the extracellular and/or intracellular spike proteins though the activation of cell signal transduction."

https://www.mdpi.com/2076-393X/9/1/36/htm

"RBD Only-Containing SARS-CoV-2 Spike Protein Does Not Elicit Cell Signaling in Human Cells

In contrast to the full-length spike protein [26,29] or the full-length SARS-CoV-2 spike protein S1 subunit [21], we found that the RBD only-containing protein (Figure 1) did not promote cell signaling..

The different effects of the full-length S1 and RBD only-containing proteins may be important considering that BNT162b2 [PFIZER commercially produced and given in phases 2 & 3 & to the public] and many other COVID-19 vaccines express the full-length spike protein, while the BNT162b1 vaccine [PFIZER phase 1] encodes only the RBD region"

Possible actions of the SARS-CoV-2 spike protein.

The SARS-CoV-2 spike protein of the intact virus targets ACE2 of the host cells to facilitate the membrane fusion and the viral entry. The SARS-CoV-2 spike protein also elicits cell signaling in human cells [21,29]. COVID-19 vaccines introduce the spike protein into the human body. In addition to eliciting an immune response that suppresses the viral entry, the spike protein produced by the COVID-19 vaccines may also affect the host cells, possibly triggering adverse events."

"Our laboratory only tested the effects of the SARS-CoV-2 spike protein in lung vascular cells and those implicated in the development of PAH [Pulmonary Arterial Hypertension]. However, this protein may also affect the cells of systemic and coronary vasculatures, eliciting other cardiovascular diseases such as coronary artery disease, systemic hypertension, and stroke. In addition to cardiovascular cells, other cells that express ACE2 have the potential to be affected by the SARS-CoV-2 spike protein, which may cause adverse pathological events. Thus, it is important to consider the possibility that the SARS-CoV-2 spike protein produced by the new COVID-19 vaccines triggers cell signaling events that promote PAH, other cardiovascular complications, and/or complications in other tissues/organs in certain individuals (Figure 3). We will need to monitor carefully the long-term consequences of COVID-19 vaccines that introduce the spike protein into the human body. Furthermore, while human data on the possible long-term consequences of spike protein-based COVID-19 vaccines will not be available soon, it is imperative that appropriate experimental animal models are employed as soon as possible to ensure that the SARS-CoV-2 spike protein does not elicit any signs of the pathogenesis of PAH or any other chronic pathological conditions."

Four years is the timeframe for developing leukaemia from the 💉

Too many unquestioning people believe that the vaccine and vaccine passport is the only way out of lockdown. They have been lulled into a mental state of learned helplessness and false hope after receiving months of fearmongering via MSM as well as enduring months of NPI‘s.
Now they want nothing further to get in the way of an exit strategy and think nothing of getting a quick injection or 2 as a way of finally getting out.

Desperate times call for desperate measures.

„Anti-vaxxers“ are being demonised by MSM as holding up herd immunity and potentially delaying reopening up (in lockdown and closed border countries).

In parts of Germany, people have not seen loved ones from other households since 2 nov, they cannot travel more than 15 km for leisure and anyway, they can only leave for essential reasons only.
Life is hell. A vax looks like a minor inconvenience in exchange for opening up.

That is their argument.

That is why the vax campaign is being welcomed by so many .

As a friend said recently, only a shock might snap them out of this trance of false hope. What kind of shock , my friend did not know.

I think you are right, Robin.
Discrediting the vax for HEALTHY people is the key .

@Robin do you have any contacts to filmmakers in Uruguay? We need some help for the rally on march 20th 🤠

Bread and Circuses: Who’s Behind the Oxford Vaccine for COVID-19? – Architects for Social Housing (ASH)
https://architectsforsocialhousing.co.uk/2020/11/25/bread-and-circuses-whos-behind-the-oxford-vaccine-for-covid-19/amp/?__twitter_impression=true

"Researchers at the Sloan Kettering Institute have found that changes in an information-carrying molecule called messenger RNA can inactivate tumor-suppressing proteins and thereby promote cancer."

In a Twist, Scientists Find Cancer Drivers Hiding in RNA, Not DNA | Memorial Sloan Kettering Cancer Center
https://www.mskcc.org/news/scientists-find-cancer-drivers-hiding-rna-not-dna?amp&__twitter_impression=true

ADENOVIRUSES ARE USED IN ASTRAZENECA-OXFORD AND SPUTNIK V.

Adenovirus-platelet interaction in blood causes virus sequestration to the reticuloendothelial system of the liver

"Intravenous (i.v.) delivery of recombinant adenovirus serotype 5 (Ad5) vectors for gene therapy is hindered by safety and efficacy problems. We have discovered a new pathway involved in unspecific Ad5 sequestration and degradation. After i.v. administration, Ad5 rapidly binds to circulating platelets, which causes their activation/aggregation and subsequent entrapment in liver sinusoids. Virus-platelet aggregates are taken up by Kupffer cells and degraded. Ad sequestration in organs can be reduced by platelet depletion prior to vector injection. Identification of this new sequestration mechanism and construction of vectors that avoid it could improve levels of target cell transduction at lower vector doses."

https://pubmed.ncbi.nlm.nih.gov/17301138/

Aaron Walton is welcome on this channel. https://twitter.com/Haydeano1/status/1371496568671588356?s=19

https://www.pei.de/EN/newsroom/hp-news/2021/210315-pei-informs-temporary-suspension-vaccination-astra-zeneca.html

...Compared to the status on 11 March 2021, additional cases (as of Monday, 15 March 2021) have now been reported in Germany. Analysing the new data status, the experts of the Paul-Ehrlich-Institut now see a striking accumulation of a special form of very rare cerebral vein thrombosis (sinus vein thrombosis) in connection with a deficiency of blood platelets (thrombocytopenia) and bleeding in temporal proximity to vaccinations with the COVID-19 vaccine AstraZeneca.

Thank you, Robin!
As usual, our complicit media do not trouble their audiences with anything useful.
My sincere hope is that the emerging reality - which I and others warned about last year - that there’s no such thing as a zero unwanted effect medical treatment, will prompt leaders to re-evaluate their cavalier approach to vaccination.
Those under 60 & in good health are not at elevated risk from this virus. It makes no sense from a medical ethics standpoint even to offer or encourage this majority group to accept an experimental intervention.
Especially when it’s unclear who gets the live round in the Russian roulette gamble.

This alone must kill stone dead the malign idiocy that is vaccine passports.

Best wishes,
Mike