Robin Monotti
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All cause mortality in the UK
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The UK digital identity and attributes trust framework - GOV.UK

The government is inviting feedback on new requirements for organisations wanting to provide or consume digital identity products and services.

Published 11 February 2021
https://www.gov.uk/government/publications/the-uk-digital-identity-and-attributes-trust-framework
"Government sets out new plans to help build trust in use of digital identities

The government has today published its draft rules of the road for governing the future use of digital identities.

Published 11 February 2021"
https://www.gov.uk/government/news/government-sets-out-new-plans-to-help-build-trust-in-use-of-digital-identities
Channel name was changed to «Robin Monotti Graziadei»
Winter Solstice ❄ was 21st December: the shortest day of the year = lowest amount of vitamin D produced via ☀️ & skin pigmentation. 2 weeks after ❄ solstice Covid19 "cases" fell off a cliff face. This means we have herd immunity & the year's seasonal infection peak is behind us
https://twitter.com/MartinKulldorff/status/1363536045200072710?s=19
Heroes of the pandemic
-Didier Raoult
-Anders Tegnell
-Johann Giesecke
-John Ioannidis
-Dr Reiner Fuellmich
-Robert Kennedy Jr
-Sucharit Bhakdi
-Martin Kulldorf
-Sunetra Gupta
-Jay Battacharya
-Vladimir Zelenko
-Mike Yeadon
-Pierre Kory
-Simone Gold
-Stella Immanuel
-Carrie Madej
And many many more. Thank you to all of you. This is just the beginning of the Great Awakening..
"In the case of COVID-19, killer T cells could mean the difference between a mild infection and a severe one that requires hospital treatment, says Annika Karlsson, an immunologist at the Karolinska Institute in Stockholm. “If they are able to kill the virus-infected cells before they spread from the upper respiratory tract, it will influence how sick you feel,” she says. They could also reduce transmission by restricting the amount of virus circulating in an infected person, meaning that the person sheds fewer virus particles into the community.

T cells could also be more resistant than antibodies to threats posed by emerging variants. Studies by Sette and his colleagues have shown that people who have been infected with SARS-CoV-2 typically generate T cells that target at least 15–20 different fragments of coronavirus proteins1. But which protein snippets are used as targets can vary widely from person to person, meaning that a population will generate a large variety of T cells that could snare a virus. “That makes it very hard for the virus to mutate to escape cell recognition,” says Sette, “unlike the situation for antibodies.”
So when laboratory tests showed that the 501Y.V2 variant identified in South Africa (also called B.1.351) is partially resistant to antibodies raised against previous coronavirus variants, researchers wondered whether T cells could be less vulnerable to its mutations.
Early results suggest that this might be the case. In a preprint published on 9 February, researchers found that most T-cell responses to coronavirus vaccination or previous infection do not target regions that were mutated in two recently discovered variants, including 501Y.V22. Sette says that his group also has preliminary evidence that the vast majority of T-cell responses are unlikely to be affected by the mutations."
https://www.nature.com/articles/d41586-021-00367-7